Cold Laser Therapy

Therapeutic Optical Windows

"Our earliest research revealed and later clinical studies confirmed that high immediate and short-term pain reliefs are extremely important for good outcome of the entire course of treatment. "
Dr. Norman Salansky, PhD
LEP2000™
Photonic Therapy System
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Facts about Laser Therapy

IMI Healing Technologies' prime mission is to bring the recognition and use by medical communities in North America and around the Globe of its LEP2000™ Photonic and Laser Therapy System, and to offer high-efficacy therapeutic devices based on scientifically and clinically proven healing properties of multi-modality photonic and laser therapy approaches.
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Therapeutic Optical Windows

Novel Concept of Pain Relief and Tissue Healing

A large body of research in cell culture and animal studies suggest that coherent or non-coherent monochromatic light with specific optical parameters can improve or reverse to normal abnormal cell and tissue processes to a normal state. These are so-called low level, low power, low intensity laser biostimulation and photobiomodulation phenomena. These phenomena are extremely sensitive to optical parameters (wavelength, power, irradiance, fluence (dose), modulation frequency, etc.). Depending on the optical parameters either a positive photo effect, an inhibition, or no effect at all is observed.

As a result of 25 years of basic and clinical research supported by the Canadian Government, Doctors Norman and Natasha Salansky discovered and optimized so-called “Therapeutic Optical Windows” (TOWs) that help to improve or reverse to normal pathological tissue conditions like tissue ischemia (depletion of cellular energy ATP), impaired microcirculation, chronic or acute inflammation, etc. TOWs represent sets of optical parameters {wavelength (nm), power (mW), irradiance (mW/cm2), fluence (dose, J/cm2), modulation frequency (Hz), duty cycle (%), etc.) and three-dimensional (3D) photon distribution within the tissue that are required to improve a particular tissue pathological condition.

In order to improve or reverse to normal a particular pathological condition of tissue (tissue ischemia, impaired microcirculation, acute or chronic inflammation, etc.) a single or several specific TOWs are required. In order to treat with high efficacy a particular disease (e.g., carpal tunnel syndrome) usually several specific TOWs are required because, in most cases, a disease is represented by several pathological tissue’s conditions.

For example, a patient with carpal tunnel syndrome may present with the following tissue pathologies (or some of them): tissue and nerve ischemia and impaired microcirculation in the carpal tunnel, chronic inflammation in surrounding tissues, tendinitis or tenosynovitis, ischemic or inflammatory swelling in hands, myofascial pain, tender and trigger points, etc. Each one of these pathological conditions may require a specific TOW.

The following are basic concepts that being implemented in concert allow substantial therapeutic effects to be induced by Low Energy Photonic and Laser Therapy (Low Level Laser Therapy, Low Energy Laser Therapy, and Low Intensity Laser Therapy):
  1. The concept of “Therapeutic Optical Windows”: the full set of optical parameters and an appropriate 3D photon distribution has to conform entirely to corresponding optical window in order to induce a desired photo-effect. If even one of the optical parameters is out of the optical window, the photo-effect could not be induced.
  2. An appropriate 3D light distribution has to be provided in the target tissue. There are incident and internal optical windows. Incident optical windows correspond to the incident optical parameters. Internal optical windows are the actual optical parameters that are “seen” by the tissue cells. Incident and internal optical windows are the same for monolayers of cell cultures.
  3. Specificity of TOWs to the desired biological mechanisms to be induced: different TOWs have to be used to induce different biological mechanisms.
  4. Timing and specificity of TOWs to the specific photo-induced mechanisms has to be taken into account for optimization of healing by monochromatic light.
  5. In particular, timing and specificity of TOWs to the phase of wound repair: substantially different TOWs are to be used at the inflammatory phase of soft tissue (wound) healing as opposed to the repair phase.
  6. A key mechanism/s induced by monochromatic light in tissue has to correspond to soft tissue pathology.
  7. Orchestration and synchronization of photo-induced phenomena at different levels of biological structures: biomolecules (e.g., ATP, DNA, RNA, gene expression), cell organelles (e.g., membrane transport), cells, tissue (e.g., vasoactive phenomena), systemic (e.g., immune system response) is required for optimal photo induced phenomena.
  8. In particular, activation of cellular metabolism has to be supported by an adequate blood supply to the target area. Specific TOWs are to be used to increase blood supply to the target area via photo-induced vasodilatation, anastomosis opening, or neovascularization/angiogenesis.
  9. A synergy between several TOWs could substantially enhance desirable therapeutic effect/ soft tissue healing.
A violation of any of the above principles results in failure to produce results in vivo, marginal or sub-optimal results (on the tales of the therapeutic optical windows). The above conceptual approach is a novel approach in medicine. It is entirely different from an allopathic (pharmaceutical) paradigm of abolishing symptoms targeting a single key molecule (enzyme, DNA, cellular messenger, etc.). Targeting with a “magic bullet” in allopathic medicine often results in distortion of metabolism and side effects. Instead, the above approach requires orchestration of numerous vital cellular and tissue processes and respect to “cell intelligence” - their ability to communicate to each other and perform quality healing.

A description of various pathological conditions of tissue could be found in the text books for medical schools Cotran RS. Robbins pathologic basis of disease. 6th ed./Cotran RS, Kumar V, Collins T. W.B. Saunders Company, 1999.

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